Some New Science about Long Haul? Zombie Viral Pieces Hang Around for a Long Time!

#93 2024 The Rowen Report

Dear Subscriber,

Some more information is emerging about chronic COVID. This arrived in one of my chat groups. It seems that some people will be marked for chronic COVID as some people produce an intense immune response called a cytokine storm that can lead to damage most anywhere in te boy. It is an overactive immune response and the bombs your immune cells hurl at the foreign stuff might actually cause collateral damage to your own cells. Some people further will retain “zombie” fragments of the virus in their bodies, leading to this continuing destructive process.

Usually, once your immune system is primed and goes after an invader, your system wins. But in the case of COVID, Gerard Wong, corresponding author of the research, and a professor in microbiology, immunology and molecular genetics at the University of California-Los Angeles, told Salon in an interview: “And, that’s the zombie part — after you destroy this virus that should be dead, it's not. It actually can reassemble into these complexes that can have very strong immune activation ability.” This is frightening on two levels. First, that viral pieces can reassemble into really hot damaging stuff, and that the particles still exist, even if the whole virus has cleared. I imaging COVID vax would cause some long haul problems the same way. The vax induces your body to make spike protein by hijacking your protein synthesis. And with the RNA being synthetic, it can last a long, long time in your system, much like synthetic (plastic) materials do on our planet.

The good news is that most of my buddies report outstanding results using ozone therapy for long haul, as do we. Better yet, get ozone quickly and hopefully you won’t have a problem with “long haul” disease or vax.

To Your Excellent Health!

Robert Jay Rowen, MD

https://www.salon.com/2024/02/02/zombie-19-fragments-linger-for-some-causing-severe-disease-outcomes-new-research-reveals/

"Zombie" COVID-19 fragments linger for some, causing severe disease outcomes, new research reveals

A new finding could be the reason why some people have long COVID or die from SARS-CoV-2

hough the COVID-19 pandemic has largely faded from the public mind, infections are still very common and not always benign like the flu. There is a link between inflammation, the immune system and COVID-19 that is not yet quite understood — and it could still be causing thousands of deaths and disabilities. 

As Salon has previously reported, severe cases of COVID can trigger a hyperinflammatory response called a "cytokine storm" so intense that it seems to exhaust the T cells critical to immune function and decrease their number. There has been evidence to suggest this can impact the immune system's ability to fight future infections from both COVID-19 and other diseases as well. More recently, researchers found that the inflammation caused by COVID-19 in pregnant women led to an inflammatory response in their infants causing respiratory distress after birth.

There is something specific about a SARS-CoV-2 infection, the virus that causes COVID, and how it can sometimes lead to an overactive immune response. Yet the way in which this occurs remains a mystery.

But now, researchers at the University of California-Los Angeles say they are one step closer to understanding how COVID-19 can result sometimes in severe outcomes or death while other coronaviruses like the common colds don’t. The answer, they believe, lies in so-called “zombie” virus fragments that linger and cause inflammation long after the virus has been destroyed. 

"After you destroy this virus that should be dead, it's not."

“There's a kind of biomimicry going on here, that once the virus is broken down these pieces are actually active and they can assemble with double-stranded RNA that's commonly found in viral infections,” Gerard Wong, corresponding author and a professor in microbiology, immunology and molecular genetics at the University of California-Los Angeles, told Salon in an interview. “And, that’s the zombie part — after you destroy this virus that should be dead, it's not. It actually can reassemble into these complexes that can have very strong immune activation ability.”

As Wong alluded to, once a virus interacts with the human immune system, the virus is usually broken down to a point where the host and its immune system wins. As a result, the immune system learns from that interaction and builds immunity to that virus by knowing exactly how to defeat it next time. But it doesn’t exactly work that way with SARS-CoV-2.