Learning in Brazil. The crucial Vitamin A connection to copper and iron metabolism.
#27 2023 The Rowen Premium Report
Dear Subscriber,
As I write this, Terri and I are on board a plane from Brazil returning to the USA. We went to South America where I presented to a large integrative healing forum on ozone therapy. We were very well received and have been asked to return next year where they plan the forum to be in Amazonia. This year it was Sao Paulo. We got a real treat also being able to visit Iguazo Falls, one of the “seven wonders” of the natural world. Here is a photo just a portion of the falls to include Terri and me, and one of me before the “Redeemer” in Rio. The photo of the falls is just a portion. We got to walk on “catwalks” above and below the falls, simply amazing.
At the medical meeting, I attended a lecture by a dentist well-heeled in nutrition, Michael Gossweller, DDS, of Indianapolis. Those of you in the upper Midwest are blessed to have this man in your neighborhood. He is wise to some of the matters I’ve written about here – copper and iron (and dental interference fields like root canals and cavitations).
I’ve told subscribers that copper is absolutely essential to control iron and keep it chaperoned. Iron is essential for our lives. It transports iron in your red cells. It participates in the electron transport chain in mitochondria. However, it is extremely reactive, especially in its Fe+2 state. It can release an electron to other molecules, including H2O2, or mitochondrial molecules to created highly toxic free radicals. In fact, iron accumulation is a major cause of aging. Women live longer than men because of monthly iron shedding with menstruation. After menopause, their death rate quickly approaches that of men.
Our bodies go to great lengths to shield tissues from iron reactive damage and has made a hose of protein chaperones. Ceruloplasmin is a little known (in conventional medicine) protein made in your liver. It carries and is activated by the mineral copper (which I’ve also written about). Ceruloplasmin is known in conventional medicine for its relation to copper, but an extremely important function is chaperoning iron to where it is supposed to go. Low ceruloplasmin activity > increased free iron, which is highly damaging to cellular components.
Here is where it gets interesting on a biochemical matter that I wasn’t aware of. One can be anemic with the appearance of iron deficiency, when the matter is not iron deficiency at all, but a deficiency of ceruloplasmin/copper. We have seen unexplained anemia that could be secondary to this effect: